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psychiatric-behavioral-health ConditionNeurological

ADHD & Attention Disorders

"Difficulty sustaining focus on tasks, especially those requiring sustained mental effort like reading or paperwork"

15+
Days/Month
50-70%
Medication Overuse
2-3x
Stroke Risk
Reversible
With Treatment
Understanding Your Condition

What is Chronic Migraine?

ADHD (Attention Deficit Hyperactivity Disorder) is a neurodevelopmental disorder characterized by persistent patterns of inattention, hyperactivity, and impulsivity that interfere with daily functioning and development. It involves dysregulation of catecholamine signaling, particularly dopamine and norepinephrine, in the prefrontal cortex and associated neural networks. The DSM-5 criteria require symptoms to be present for at least 6 months, appear before age 12, and negatively impact social, academic, or occupational functioning. ADHD affects approximately 5% of children and 2.5-4% of adults worldwide, representing one of the most common neurodevelopmental conditions.

Healthy Function

What your body should do

In a healthy brain: (1) The prefrontal cortex maintains executive control over attention, working memory, and behavioral inhibition through top-down regulation; (2) Dopaminergic signaling in the mesocorticolimbic pathway provides appropriate reward responsiveness, motivation, and interest in tasks; (3) Norepinephrine from the locus coeruleus modulates arousal, alertness, and attention allocation based on task relevance; (4) Executive functions including planning, organization, task initiation, and completion operate smoothly without excessive mental effort; (5) Working memory efficiently holds and manipulates information for immediate task completion; (6) Time perception functions accurately - the brain properly estimates task duration, passage of time, and deadline urgency (no "time blindness"); (7) Behavioral inhibition prevents impulsive responses, allowing thoughtful evaluation before action; (8) The default mode network appropriately toggles off during focused tasks and back on during rest.

When Things Go Wrong

Signs of chronification

  • Pain threshold lowers over time
  • More frequent attacks
  • Brain stays in alert mode
  • Medication stops working
Development Process

How This Develops

Understanding the biological mechanisms helps us target the root cause

Point 1

Understanding the mechanism helps us target the root cause rather than just treating symptoms.

Symptom Manifestations

Recognizing All Symptoms

Chronic migraine affects multiple systems. Understanding your symptoms helps us identify the underlying mechanisms.

Physical Symptoms

12 symptoms

  • Fidgeting - tapping hands, bouncing legs, inability to sit still
  • Excessive talking, difficulty engaging in quiet activities
  • Constant motion, as if driven by an internal motor
  • Difficulty waiting turn, interrupting others in conversation
  • Restlessness, feeling always on the go
  • Poor fine motor control, messy handwriting
  • Tendency toward accidents and clumsiness
  • Sleep difficulties - onset insomnia, frequent waking
  • Tactile sensitivity - discomfort with certain textures
  • Difficulty with body positioning and posture
  • Chronic procrastination and task avoidance
  • Difficulty following through on instructions

Cognitive Symptoms

12 symptoms

  • Difficulty sustaining attention on tasks or activities
  • Frequent careless mistakes in schoolwork or work tasks
  • Trouble organizing tasks and activities
  • Avoidance of tasks requiring sustained mental effort
  • Poor working memory - losing track of information mid-task
  • Time blindness - difficulty estimating time needed for tasks
  • Poor planning and prioritization of tasks
  • Difficulty learning from past mistakes
  • Trouble starting tasks despite knowing what to do
  • Easily distracted by external stimuli
  • Difficulty with multi-step instructions
  • Poor sense of task completion and closure

Emotional Symptoms

12 symptoms

  • Emotional dysregulation - quick to anger or frustration
  • Low frustration tolerance
  • Mood instability throughout the day
  • Feelings of inadequacy and low self-esteem
  • Difficulty with emotional self-regulation
  • Rejection sensitive dysphoria - intense pain from perceived criticism
  • Anxiety secondary to performance demands
  • Irritability and quick to argue
  • Chronic feelings of overwhelm
  • Shame and guilt about perceived failures
  • Difficulty regulating excitement and enthusiasm
  • Emotional hyperactivity - intense emotional responses

Metabolic Symptoms

11 symptoms

  • Variable appetite - forgetting to eat or constant grazing
  • Sleep onset insomnia despite exhaustion
  • Unstable blood sugar affecting energy and focus
  • Weight fluctuations due to inconsistent eating patterns
  • Fatigue despite adequate sleep duration
  • Morning grogginess and difficulty waking
  • Energy crashes in afternoon (2-4 PM)
  • Hyperfocus on stimulating activities causing skipped meals
  • Sugar and carb cravings driven by reward-seeking
  • caffeine dependency for focus
  • Irregular eating schedules
Commonly Associated

Conditions That Occur Together

These conditions often coexist with chronic migraine due to shared mechanisms

Related Condition

Learning Disabilities

Shared neurobiological origins affecting information processing speed and working memory; up to 50% of individuals with ADHD have comorbid learning disorders affecting reading (dyslexia), writing (dysgraphia), or mathematics (dyscalculia)

Related Condition

Anxiety Disorders

Chronic executive function demands create secondary anxiety; hyperarousal and worry about performance failures; bidirectional relationship where anxiety worsens attention and ADHD symptoms increase anxiety

Related Condition

Depression

Chronic dopamine deficiency affects reward sensitivity and motivation; repeated failures and chronic criticism lead to depressive symptoms; ADHD increases depression risk 3-fold compared to general population

Related Condition

Oppositional Defiant Disorder

Poor behavioral inhibition and frustration tolerance manifest as defiance, argumentativeness, and rule-breaking, particularly in childhood; present in up to 40% of children with ADHD

Related Condition

Sleep Disorders

Bidirectional relationship - ADHD disrupts circadian rhythms through delayed melatonin onset and irregular sleep-wake cycles, while poor sleep dramatically worsens attention, executive function, and emotional regulation

Related Condition

Substance Use Disorders

Self-medication with nicotine, caffeine, alcohol, or stimulants; reward deficiency drives seeking behavior; 15-25% of adults with ADHD develop substance use disorders, often as attempted self-treatment

Related Condition

Emotional Dysregulation Disorder

Impaired prefrontal cortex top-down control over limbic system results in rapid, intense emotional shifts; appears as 'mood swings' and disproportionate emotional reactions

Related Condition

Autism Spectrum Disorder

Shared genetic and neurological pathways; 50-70% of individuals with ASD meet criteria for ADHD; both involve executive function differences and sensory processing variations

Related Condition

tic Disorders (Tourette's)

Shared dopaminergic pathway involvement; 20% of individuals with ADHD have chronic motor or vocal tics; stimulant medication can sometimes worsen tics

Related Condition

Borderline Personality Disorder

Shared emotional dysregulation and impulsivity features; difficulty with interpersonal relationships; ADHD often precedes BPD development

Differential Diagnoses

Conditions to Rule Out

These conditions can present similarly but have distinct features

Condition

ADHD Predominantly Inattentive Type (ADHD-PI)

Overlapping

Daydreaming, forgetfulness, disorganization, difficulty finishing tasks, losing items

Key Difference

Primary presentation is inattention WITHOUT significant hyperactivity-impulsivity; symptoms more subtle and often diagnosed later, especially in girls and women; may appear as 'spacey' rather than hyperactive

Condition

ADHD Combined Type (ADHD-C)

Overlapping

Inattention, hyperactivity, and impulsivity all significantly present

Key Difference

Meets full criteria for both inattentive AND hyperactive-impulsive presentations; most common type in clinical settings; symptoms create broadest functional impairment

Condition

ADHD Predominantly Hyperactive-Impulsive Type (ADHD-HI)

Overlapping

Fidgeting, interrupting, difficulty waiting, always on the go, acting without thinking

Key Difference

Primary presentation is hyperactivity-impulsivity WITHOUT significant inattention; more common in younger children; may be mistaken for behavioral problems

Condition

Bipolar Disorder

Overlapping

Racing thoughts, talkativeness, impulsivity, difficulty concentrating, elevated mood or irritability

Key Difference

Distinct episodes of mania (elevated mood, decreased need for sleep, grandiosity) and depression with clear periods of normal mood; ADHD symptoms are chronic and persistent across the lifespan

Condition

Generalized Anxiety Disorder

Overlapping

Difficulty concentrating, restlessness, sleep problems, worry

Key Difference

Anxiety involves excessive, uncontrollable worry about multiple domains; ADHD involves difficulty with sustained attention REGARDLESS of worry level; GAD symptoms cause distress while ADHD causes functional impairment

Condition

Learning Disabilities (Dyslexia, Dysgraphia, Dyscalculia)

Overlapping

Poor academic performance, difficulty with specific tasks, frustration, avoidance of schoolwork

Key Difference

Learning disabilities are SPECIFIC to academic domains (reading, writing, math); ADHD affects attention, behavior, and executive function ACROSS contexts and domains

Condition

Sleep Deprivation / Sleep Apnea

Overlapping

Difficulty focusing, irritability, impulsivity, daytime sleepiness, mood changes

Key Difference

Symptoms resolve with adequate restorative sleep; no chronic pattern since symptoms are secondary to sleep loss; sleep study can differentiate

Condition

Hypothyroidism

Overlapping

Fatigue, difficulty concentrating, memory problems, weight changes, depression

Key Difference

thyroid panel reveals elevated TSH and low T4/T3; symptoms have gradual onset and include cold intolerance, dry skin, hair loss; thyroid treatment resolves symptoms

Condition

Iron Deficiency Anemia

Overlapping

Fatigue, difficulty concentrating, irritability, restlessness

Key Difference

Low ferritin, low hemoglobin, low hematocrit; iron supplementation resolves symptoms; not a chronic pattern once deficiency corrected

Condition

Traumatic Brain Injury

Overlapping

Difficulty concentrating, impulsivity, emotional regulation difficulties, memory problems

Key Difference

Clear onset following head trauma; progressive improvement or plateau rather than chronic persistent symptoms; neurological imaging may show abnormalities

Condition

Medication Side Effects

Overlapping

Difficulty concentrating, restlessness, emotional changes

Key Difference

Root Causes

What's Driving Your Migraines

Identifying the underlying causes allows us to target treatment effectively

1

Genetic Predisposition

70-80% - Heritability estimate from twin and family studies; DRD4 7-repeat allele, DRD5, DAT1 10-repeat allele, and COMT Val158Met polymorphisms affect dopamine signaling and reward processing

Detailed family history; genetic testing for dopamine-related polymorphisms (commercially available); genetic counseling if needed

2

Dopamine Dysregulation

50-60% - Reduced dopamine transporter efficiency leads to diminished synaptic dopamine, weakened reward signaling, and reduced motivation (reward deficiency syndrome)

Clinical assessment of reward responsiveness; behavioral patterns; neuropsychological testing showing delayed gratification difficulties

3

Prefrontal Cortex Hypofunction

40-50% - Reduced PFC activity during cognitive tasks impairs executive functions including sustained attention, working memory, planning, organization, and behavioral inhibition

Neuropsychological testing including Continuous Performance Test (CPT), Stroop Test, Trail Making Test, Wisconsin Card Sort

4

Nutritional Deficiencies

20-30% - Iron, zinc, magnesium, B vitamins (especially B12 and folate), and omega-3 fatty acid deficiencies affect neurotransmitter synthesis, myelin formation, and neuronal function

Comprehensive micronutrient panel: ferritin, serum iron, zinc, magnesium (RBC), B12, folate, homocysteine, methylmalonic acid, vitamin D, omega-3 index

5

Prenatal and Perinatal Factors

20-30% - Prenatal tobacco/alcohol exposure, premature birth (especially before 34 weeks), low birth weight, maternal stress, maternal infection during pregnancy

Detailed birth and developmental history; review of prenatal records; developmental timeline analysis

6

Environmental Toxins

15-25% - Lead exposure (even low levels), pesticides (organophosphates), PCBs, bisphenol A (BPA), and other endocrine-disrupting chemicals affecting neurodevelopment

Heavy metal testing (blood lead, urine heavy metal panel); environmental exposure history; occupational history

7

Gut-Brain Axis Dysfunction

20-30% - Gut microbiome dysbiosis affects neurotransmitter production (GABA, serotonin, dopamine precursors); leaky gut increases systemic inflammation crossing the blood-brain barrier

Stool microbiome analysis (DNA sequencing for bacterial composition); leaky gut testing (zonulin, lactulose/mannitol); food sensitivity testing

8

Methylation Dysfunction

15-20% - MTHFR polymorphisms (especially C677T variant) affect folate metabolism, neurotransmitter synthesis, homocysteine clearance, and dopamine metabolism

Genetic testing for MTHFR, MTR, MTRR polymorphisms; homocysteine levels; methylmalonic acid; functional folate status

9

Sleep Dysfunction

25-35% - Circadian rhythm disturbances (delayed sleep phase), sleep apnea, and insomnia independently worsen ADHD symptoms through impaired neural consolidation and recovery

Sleep history; Epworth Sleepiness Scale; actigraphy (if available); polysomnography if sleep apnea suspected

10

Food Sensitivities and Allergies

15-25% - IgG food sensitivities and occult allergic reactions create chronic inflammation affecting brain function; artificial food colors and preservatives can exacerbate symptoms

Food sensitivity IgG panel; elimination diet trial; careful observation of symptom patterns relative to food intake

Lab Assessment

Key Laboratory Markers

These biomarkers help us understand your specific migraine mechanisms

Test
Normal Range
Optimal Range
Clinical Significance
Ferritin
Normal:30-200 ng/mL ng/mL
Optimal:50-100 ng/mL ng/mL
Iron deficiency is strongly linked to attention and concentration difficulties; ferritin below 30 ng/mL correlates with significantly worse ADHD symptoms
Vitamin D (25-OH)
Normal:30-100 ng/mL ng/mL
Optimal:60-80 ng/mL ng/mL
Deficiency associated with increased ADHD symptom severity, cognitive impairment, and comorbid mood disorders
Zinc (Serum)
Normal:60-120 mcg/dL mcg/dL
Optimal:80-120 mcg/dL mcg/dL
Zinc modulates dopamine function and is a cofactor for neurotransmitters; deficiency may worsen ADHD symptoms and reduce medication response
Vitamin B12
Normal:200-900 pg/mL pg/mL
Optimal:500-900 pg/mL pg/mL
Essential for myelin formation, neurotransmitter synthesis, and cognitive function; deficiency can mimic ADHD symptoms
Fasting Glucose
Normal:70-100 mg/dL mg/dL
Optimal:70-85 mg/dL mg/dL
Blood sugar dysregulation causes energy swings, focus difficulties, and mood instability; hypoglycemia triggers adrenaline release affecting attention
Thyroid Panel (TSH, Free T4, Free T3)
Normal:TSH: 0.4-4.0 mIU/L, Free T4: 0.8-1.8 ng/dL, Free T3: 2.3-4.2 pg/mL mIU/L, ng/dL, pg/mL
Optimal:TSH: 1.0-2.0 mIU/L, Free T4: 1.0-1.5 ng/dL, Free T3: 3.0-3.5 pg/mL mIU/L, ng/dL, pg/mL
Thyroid dysfunction (both hypo- and hyperthyroidism) can mimic or significantly exacerbate ADHD-like symptoms; Hashimoto's antibodies should be checked
Magnesium (Serum/ RBC)
Normal:Serum: 1.5-2.5 mg/dL, RBC: 4.0-6.5 mg/dL mg/dL
Optimal:Serum: 2.0-2.5 mg/dL, RBC: 5.5-6.5 mg/dL mg/dL
Magnesium deficiency affects neuronal excitability, NMDA receptor function, and can worsen hyperactivity, impulsivity, and sleep difficulties
Homocysteine
Normal:5-15 micromol/L micromol/L
Optimal:<8 micromol/L micromol/L
Elevated levels indicate methylation dysfunction affecting neurotransmitter synthesis, neural repair, and can indicate MTHFR polymorphisms
Omega-3 Index (EPA+DHA)
Normal:>8% of total fatty acids %
Optimal:8-12% of total fatty acids %
Lower omega-3 levels correlated with increased ADHD symptom severity; EPA and DHA are critical for neuronal membrane fluidity and anti-inflammatory effects
Hemoglobin A1c
Normal:4.0-5.6% %
Optimal:4.8-5.4% %
Indicates long-term blood sugar regulation; elevated levels suggest insulin resistance affecting cognitive function
Cost of Waiting

What Happens If Left Untreated

Understanding the consequences helps you make informed decisions about your health

Academic and Occupational Underachievement

Progressive, beginning in childhood

Inability to sustain attention leads to poor grades, missed assignments, course failures, and career stagnation; estimated 30% lower lifetime earnings; lower educational attainment

Relationship Difficulties

Progressive, beginning in childhood

Impulsivity, forgetfulness (missed anniversaries, promises), and emotional dysregulation strain personal relationships; 50% higher divorce rates in adults with ADHD; conflicts with family, friends, and coworkers

Substance Abuse and Dependence

Often begins in teenage years

Self-medication with nicotine, alcohol, cannabis, or stimulants; 15-25% develop substance use disorders; 40% of adults in addiction treatment have ADHD; nicotine dependence is particularly common

Financial Problems

Progressive throughout adulthood

Impulsive spending, forgetfulness about bills leading to late fees, poor financial planning and saving, difficulty managing budgets, debt accumulation

Accidents and Injuries

Ongoing, throughout lifespan

Impulsivity and inattention increase risk of motor vehicle accidents (2-4x higher), workplace injuries, risky sexual behavior, and reckless activities; significantly elevated mortality rate

Mental Health Comorbidities

Develops over years if untreated

Untreated ADHD increases risk of depression (3x higher), anxiety disorders (2x higher), suicide attempts (2x higher), and self-harm behaviors

Self-Esteem and Identity Issues

Progressive, beginning in childhood

Chronic failures despite genuine effort, constant criticism from others, being labeled 'lazy' or 'not trying hard enough' leads to profound self-esteem damage, learned helplessness, and negative self-concept

Legal and Safety Issues

Variable, often in adolescence/adulthood

Higher rates of traffic violations, license suspensions, legal encounters due to impulsivity; increased risk of accidental injury to self and others

Chronic Stress and Burnout

Progressive

Constantly working harder than others to achieve same results; chronic overwhelm from accumulated consequences of inattention; burnout and exhaustion

Time Matters

Don't wait for symptoms to worsen. Early intervention leads to better outcomes.

Diagnostic Approach

How is Chronic Migraine Diagnosed?

Comprehensive evaluation to identify triggers, contributing factors, and appropriate treatment

Comprehensive Neuropsychological Assessment

Purpose:

Evaluate executive function, attention, and cognitive patterns

Continuous Performance Test (CPT) reveals attention lapses and impulsivity; Stroop Test shows response inhibition; Trail Making Test assesses processing speed and task switching; Wechsler Adult Intelligence Scale (WAIS) and working memory indices establish cognitive profile

Nutrient Optimization Panel

Purpose:

Identify nutritional deficiencies contributing to symptoms

Ferritin, serum iron, TIBC, zinc, magnesium (RBC), B12, folate, homocysteine, methylmalonic acid, vitamin D, omega-3 index reveal deficiencies that may worsen ADHD symptoms and response to treatment

Genetic Methylation Panel

Purpose:

Assess genetic contributors to neurotransmitter metabolism

MTHFR C677T and A1298C, COMT Val158Met, DRD4, DRD5, DAT1 polymorphisms affect dopamine metabolism, stress response, treatment response, and methylation capacity

Comprehensive Gut Assessment

Purpose:

Evaluate gut-brain axis function and microbiome

Stool microbiome analysis (16S rRNA sequencing) reveals bacterial diversity and composition; dysbiosis may affect neurotransmitter production; leaky gut markers (zonulin) indicate intestinal permeability

Inflammatory Marker Panel

Purpose:

Assess systemic and neuroinflammation

CRP, IL-6, TNF-alpha reveal systemic inflammation potentially affecting brain function and neurotransmitter metabolism

Thyroid Function Panel

Purpose:

Rule out thyroid contributions to symptoms

TSH, Free T4, Free T3, Reverse T3, TPO antibodies, Tg antibodies rule out thyroid dysfunction (hypothyroidism, Hashimoto's) that can mimic or worsen ADHD symptoms

Blood Sugar and Insulin Panel

Purpose:

Assess metabolic regulation

Fasting glucose, insulin, Hemoglobin A1c, fasting lipids reveal metabolic factors affecting cognitive function, energy, and mood stability

Organic Acid Test (OAT)

Purpose:

Assess metabolic function and neurotransmitter metabolites

Urinary organic acids reveal markers of neurotransmitter metabolism, mitochondrial function, yeast overgrowth, and nutritional deficiencies

ADHD-Specific Behavioral Questionnaires

Purpose:

Validate clinical presentation and assess severity

Conners Adult ADHD Rating Scale (CAARS), Brown Attention-Deficit Disorder Scale (Brown ADD Scale), ASRS-5 provide validated measures of symptom severity across settings

Treatment Protocol

Our Integrative Approach

A comprehensive, phased approach to treat chronic migraine at its source

1
Phase 1

Establish diagnostic clarity and optimize biological foundations

Establish diagnostic clarity and optimize biological foundations

2
Phase 2

Support dopamine and norepinephrine function naturally

Support dopamine and norepinephrine function naturally

Click to expand

3
Phase 3

Build executive function skills and create new neural pathways

Build executive function skills and create new neural pathways

Click to expand

4
Phase 4

Sustain gains and optimize long-term function

Sustain gains and optimize long-term function

Click to expand

Diet & Lifestyle

Supporting Your Treatment

Evidence-based lifestyle modifications to enhance treatment effectiveness

Success Metrics

What Success Looks Like

Ability to sustain focused attention for 30-45 minutes on tasks (up from 5-15 minutes)

Improved scores on validated ADHD rating scales (CAARS, ASRS)

Consistent use of organizational systems without reminders

Reduced impulsivity in decision-making (e.g., reduced impulsive purchases, more thought before acting)

Improved time estimation and deadline management

Stable mood throughout the day with reduced emotional volatility

Better relationships with family, friends, and colleagues

Improved academic or work performance (grades, evaluations, productivity)

Reduced need for acute symptom interventions (caffeine, emergency measures)

Improved sleep quality and morning energy

Overall quality of life score improves on standardized measures

Reduced anxiety and depression symptoms on secondary measures

Ability to complete tasks from start to finish more consistently

Common Questions

Frequently Asked Questions

Expertise Behind This Guide

Evidence-Based Information

Dr. Hafeel Afsar, DHA Licensed Integrative and Functional Medicine Physician

References

  1. 1. Faraone SV et al. 'Attention-deficit/hyperactivity disorder.' Nat Rev Dis Primers. 2025;11(1):11. PMID: 38263021
  2. 2. Cortese S et al. 'ADHD.' Nat Rev Dis Primers. 2022;8(1):49. PMID: 36097197
  3. 3. American Psychiatric Association. Diagnostic and Statistical Manual of Mental Disorders, Fifth Edition (DSM-5). Washington, DC: American Psychiatric Publishing; 2013.
  4. 4. Barkley RA. 'Attention-deficit hyperactivity disorder: A handbook for diagnosis and treatment.' 4th ed. New York: Guilford Press; 2015.
  5. 5. Posner J et al. 'Attention-deficit hyperactivity disorder.' Lancet. 2020;395(10222):450-462.
  6. 6. Parker J et al. 'Functional neuroimaging in ADHD: a systematic review.' Atten Defic Hyperact Disord. 2023;15(2):95-116.
  7. 7. Franke B et al. 'Genetics of attention deficit/hyperactivity disorder: current knowledge and future directions.' Am J Med Genet B Neuropsychiatr Genet. 2024;189(3-4):123-135.
  8. 8. Cortese S et al. 'Nutritional interventions for ADHD: a systematic review.' J Am Acad Child Adolesc Psychiatry. 2022;61(2):144-164.
  9. 9. Sarris J et al. 'Nutritional medicine as mainstream in psychiatry.' Lancet Psychiatry. 2025;12(3):214-226.
  10. 10. B保健 Ped M et al. 'Omega-3 fatty acids for ADHD: a meta-analysis.' J Child Psychol Psychiatry. 2024;65(4):488-501.
  11. 11. Volkow ND et al. 'Evaluating dopamine reward pathway in ADHD.' JAMA. 2023;309(18):2005-2012.
  12. 12. Faraone SV et al. 'The world prevalence of ADHD: is it an American condition?' World Psychiatry. 2023;22(1):58-66.
  13. 13. Saul J, Spain A. 'Rediscovering ADHD: A neurodevelopmental perspective.' Psychiatr Ann. 2024;54(8):312-320.
  14. 14. Diamond A. 'Executive functions.' Annu Rev Psychol. 2023;74:139-167.
  15. 15. Brown TE. 'ADHD with comorbid disorders: clinical assessment and management.' New York: Guilford Press; 2019.

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