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psychiatric-behavioral-health ConditionNeurological

Anxiety Disorders

"Racing thoughts and constant worry that feels impossible to control"

15+
Days/Month
50-70%
Medication Overuse
2-3x
Stroke Risk
Reversible
With Treatment
Understanding Your Condition

What is Chronic Migraine?

Anxiety Disorders are a group of mental health conditions characterized by persistent, excessive fear and worry that interfere with daily functioning. They involve dysregulation of the amygdala, hyperactivity of the HPA axis, and neurotransmitter imbalances in serotonin, norepinephrine, and GABA systems. Common manifestations include generalized anxiety disorder (GAD), panic disorder, social anxiety disorder, and specific phobias.

Healthy Function

What your body should do

A healthy stress response system involves the amygdala accurately detecting threats, the HPA axis appropriately activating the fight-or-flight response, and parasympathetic rebound to return to homeostasis. Normal anxiety serves as a protective mechanism, triggering alertness and focus before important events. The prefrontal cortex effectively regulates emotional responses, GABA produces calming effects, serotonin maintains mood stability, and cortisol follows a healthy diurnal rhythm with morning peaks and evening troughs.

When Things Go Wrong

Signs of chronification

  • Pain threshold lowers over time
  • More frequent attacks
  • Brain stays in alert mode
  • Medication stops working
Development Process

How This Develops

Understanding the biological mechanisms helps us target the root cause

Point 1

Understanding the mechanism helps us target the root cause rather than just treating symptoms.

Symptom Manifestations

Recognizing All Symptoms

Chronic migraine affects multiple systems. Understanding your symptoms helps us identify the underlying mechanisms.

Physical Symptoms

10 symptoms

  • Racing heart, palpitations, and chest tightness
  • Shortness of breath and feeling like you cannot get enough air
  • Trembling, shaking, and muscle tension
  • Sweating profusely, especially on palms and forehead
  • Digestive issues: nausea, IBS flare-ups, diarrhea
  • Headaches, often tension-type
  • Fatigue despite adequate sleep
  • Insomnia or difficulty staying asleep
  • Dizziness and lightheadedness
  • Hot flashes and chills

Cognitive Symptoms

10 symptoms

  • Intrusive, racing thoughts that won't stop
  • Catastrophic thinking and worst-case scenario prediction
  • Difficulty concentrating and mind going blank
  • Memory problems, especially under stress
  • Decision paralysis and overthinking
  • Constant worry about future events
  • Rumination on past experiences
  • Sense of unreality or depersonalization
  • Fear of losing control or going crazy
  • Anticipatory anxiety about upcoming events

Emotional Symptoms

10 symptoms

  • Persistent feelings of dread and impending doom
  • Irritability and short temper
  • Feeling on edge and jumpy
  • Overwhelming fear without clear trigger
  • Emotional numbness or feeling detached
  • Frustration with inability to control worry
  • Shame about anxiety symptoms
  • Fear of judgment by others
  • Panic attacks with intense fear peaks
  • Social withdrawal and isolation

Metabolic Symptoms

8 symptoms

  • Weight changes from stress eating or loss of appetite
  • Caffeine sensitivity and jitters
  • Blood sugar crashes causing anxiety spikes
  • Hormonal fluctuations affecting anxiety levels
  • Energy crashes throughout the day
  • Adrenal fatigue from chronic stress
  • Thyroid dysfunction secondary to chronic anxiety
  • Increased inflammation markers
Commonly Associated

Conditions That Occur Together

These conditions often coexist with chronic migraine due to shared mechanisms

Related Condition

Depression

Bidirectional relationship exists; serotonin and norepinephrine dysregulation affects both mood systems, with up to 50% of anxiety disorder patients meeting criteria for comorbid depression

Related Condition

Gut-Brain Axis Dysfunction

The vagus nerve transmits gut inflammation to the brain; altered gut microbiome reduces GABA and serotonin production, directly impacting anxiety regulation

Related Condition

Thyroid Disorders

Hyperthyroidism mimics anxiety symptoms through excess thyroid hormone increasing adrenergic sensitivity; Hashimoto's autoimmune activity also triggers anxiety through inflammation

Related Condition

SIBO (Small Intestinal Bacterial Overgrowth)

Bacterial overproduction of hydrogen and methane triggers the gut-brain axis, causing anxiety symptoms; treating SIBO often reduces anxiety significantly

Related Condition

Blood Sugar Dysregulation

Hypoglycemia triggers adrenaline release as a counter-regulatory hormone, producing anxiety-like symptoms including shakiness, sweating, and panic

Related Condition

Adrenal Fatigue

Chronic stress depletes adrenal function, leading to dysregulated cortisol patterns that disrupt sleep, mood stability, and anxiety regulation

Related Condition

Sleep Disorders

Sleep deprivation increases amygdala reactivity while reducing prefrontal cortex regulation, creating a neurobiological vulnerability to anxiety

Related Condition

Nutrient Deficiencies

Deficiencies in B vitamins, magnesium, zinc, and omega-3 fatty acids impair neurotransmitter synthesis and neuronal function, exacerbating anxiety

Differential Diagnoses

Conditions to Rule Out

These conditions can present similarly but have distinct features

Condition

Generalized Anxiety Disorder (GAD)

Overlapping

Excessive worry, difficulty sleeping, muscle tension

Key Difference

Worry is diffuse and unfocused on specific situations; present for at least 6 months; diagnosis requires 3+ of: restlessness, fatigue, difficulty concentrating, irritability, muscle tension, sleep disturbance

Condition

Panic Disorder

Overlapping

Palpitations, shortness of breath, sweating, sense of doom

Key Difference

Characterized by discrete panic attacks with abrupt onset and peak within minutes; concern about additional attacks leads to behavioral changes

Condition

Social Anxiety Disorder

Overlapping

Anticipatory worry, physical symptoms in social situations

Key Difference

Fear specifically centers on social scrutiny and potential embarrassment; avoidance of social situations is prominent

Condition

Separation Anxiety Disorder

Overlapping

Excessive worry when away from attachment figures

Key Difference

Fear centers on separation from home or loved ones; typically begins in childhood but can persist into adulthood

Condition

OCD (Obsessive-Compulsive Disorder)

Overlapping

Intrusive thoughts, ritualized behaviors to reduce anxiety

Key Difference

Characterized by obsessions (recurrent thoughts) and compulsions (rituals) performed to neutralize obsessions; insight is often impaired

Condition

PTSD

Overlapping

Hypervigilance, intrusive memories, avoidance

Key Difference

Anxiety stems from specific traumatic event; presence of re-experiencing phenomena, negative alterations in cognition and mood

Condition

Hyperthyroidism

Overlapping

Anxiety, irritability, palpitations, weight loss, insomnia

Key Difference

Positive thyroid function tests; additional symptoms: heat intolerance, tremor, exophthalmos, goiter

Condition

General Medical Conditions

Overlapping

Anxiety symptoms secondary to physiological causes

Key Difference

Anxiety is directly caused by medical condition (pheochromocytoma, arrhythmias, vestibular disorders); resolves with treatment of underlying condition

Root Causes

What's Driving Your Migraines

Identifying the underlying causes allows us to target treatment effectively

1

Genetic Predisposition

30-40% - Family history increases risk 2-5x; specific genes identified in serotonin transport and COMT enzyme function

Family history assessment; genetic testing for anxiety-related polymorphisms (5-HTTLPR, COMT)

2

HPA Axis Dysregulation

35% - Chronic stress leads to sustained cortisol elevation and eventual adrenal exhaustion

Cortisol testing (morning, evening, and optionally diurnal curve); DHEA-S levels; ACTH stimulation test

3

Neurotransmitter Imbalance

30% - Reduced GABA, serotonin, and/or elevated norepinephrine disrupts calmAlertness balance

Comprehensive neurotransmitter panel; consider trial of GABA-supporting supplements

4

Amygdala Hyperactivity

25% - The fear center overresponds to neutral stimuli, triggering fight-or-flight unnecessarily

fMRI neuroimaging (research setting); clinical observation of trigger response patterns

5

Inflammatory Processes

20% - Elevated cytokines (IL-6, TNF-alpha) cross the blood-brain barrier and affect mood centers

Inflammatory markers: CRP, IL-6, TNF-alpha; treat underlying inflammation

6

Gut Microbiome Dysbiosis

25% - Altered gut bacteria reduce production of anxiety-calming neurotransmitters

Stool microbiome analysis; SIBO breath testing; treat dysbiosis and observe anxiety changes

7

Nutrient Deficiencies

20% - B vitamins, magnesium, zinc, and omega-3s are essential for neurotransmitter synthesis

Comprehensive micronutrient panel; correct deficiencies through diet and supplementation

8

Early Life Stress and Attachment

25% - Childhood adversity alters stress response system development and attachment patterns

Comprehensive history; ACE (Adverse Childhood Experiences) score; attachment style assessment

9

Cognitive Patterns

30% - Anxiety sensitivity, catastrophic thinking, and intolerance of uncertainty maintain anxiety

Validated questionnaires: ASI (Anxiety Sensitivity Index), BCQ (Breathing Confidence Questionnaire)

Lab Assessment

Key Laboratory Markers

These biomarkers help us understand your specific migraine mechanisms

Test
Normal Range
Optimal Range
Clinical Significance
Cortisol (Morning)
Normal:5-25 mcg/dL mcg/dL
Optimal:8-14 mcg/dL mcg/dL
HPA axis function; elevated levels indicate chronic stress response
Cortisol (Evening)
Normal:<10 mcg/dL mcg/dL
Optimal:<5 mcg/dL mcg/dL
Healthy diurnal rhythm; elevated evening cortisol disrupts sleep
DHEA-S
Normal:150-350 mcg/dL mcg/dL
Optimal:200-300 mcg/dL mcg/dL
Adrenal hormone; low levels suggest adrenal fatigue
Serotonin
Normal:50-200 ng/mL ng/mL
Optimal:100-150 ng/mL ng/mL
Mood regulation; deficiency linked to anxiety and depression
GABA
Normal:200-400 pmol/mL pmol/mL
Optimal:280-350 pmol/mL pmol/mL
Primary inhibitory neurotransmitter; deficiency fails to calm the nervous system
Vitamin D
Normal:30-100 ng/mL ng/mL
Optimal:50-70 ng/mL ng/mL
Immune and neurological function; deficiency associated with anxiety
Magnesium
Normal:1.5-2.5 mg/dL mg/dL
Optimal:2.0-2.3 mg/dL mg/dL
Nervous system relaxation; deficiency exacerbates anxiety symptoms
B12
Normal:200-900 pg/mL pg/mL
Optimal:500-800 pg/mL pg/mL
Neurological function; deficiency can mimic anxiety symptoms
TSH
Normal:0.4-4.0 mIU/L mIU/L
Optimal:1.0-2.0 mIU/L mIU/L
Thyroid function; hyperthyroidism can present as anxiety
Blood Glucose (Fasting)
Normal:70-100 mg/dL mg/dL
Optimal:75-85 mg/dL mg/dL
Blood sugar stability; hypoglycemia can trigger anxiety symptoms
Cost of Waiting

What Happens If Left Untreated

Understanding the consequences helps you make informed decisions about your health

Chronic Physical Health Deterioration

Years

Prolonged cortisol elevation weakens immune function, accelerates cardiovascular disease, contributes to metabolic syndrome, and promotes premature aging

Depression Development

Months to years

Up to 60% of untreated anxiety disorder patients develop major depressive disorder; neurobiological overlap makes prevention critical

Substance Abuse and Dependence

Often within first year

Self-medication with alcohol, benzodiazepines, or other substances leads to dependence; dual diagnosis complicates treatment significantly

Social and Occupational Impairment

Progressive

Avoidance behaviors expand to limit social interactions and career opportunities; relationships suffer, career advancement stalls

Physical Symptom Exacerbation

Ongoing

Chronic anxiety manifests as IBS, chronic pain, cardiovascular symptoms, and immune dysfunction; treating anxiety often resolves these

Cognitive Decline

Years

Chronic stress and cortisol exposure damage hippocampal neurons, affecting memory and cognitive function

Quality of Life Degradation

Immediate and progressive

Constant worry and physical symptoms prevent enjoyment of life; diminished capacity for happiness and fulfillment

Time Matters

Don't wait for symptoms to worsen. Early intervention leads to better outcomes.

Diagnostic Approach

How is Chronic Migraine Diagnosed?

Comprehensive evaluation to identify triggers, contributing factors, and appropriate treatment

Comprehensive Blood Panel

Purpose:

Rule out medical causes and assess nutritional status

CBC, CMP, TSH, cortisol (AM/PM), DHEA-S, vitamin D, B12, magnesium, inflammatory markers

Neurotransmitter Panel

Purpose:

Assess GABA, serotonin, norepinephrine, and dopamine levels

Urinary neurotransmitter levels reflecting central nervous system status

Adrenal Stress Index

Purpose:

Evaluate HPA axis function and cortisol rhythm

Cortisol levels at 4 points throughout the day plus DHEA; reveals dysregulation patterns

Stool Microbiome Analysis

Purpose:

Assess gut-brain axis influence on anxiety

Bacterial diversity, pathogenic organisms, SIBO markers, leaky gut indicators

Food Sensitivity Panel

Purpose:

Identify inflammatory food triggers affecting brain function

IgG and IgA reactions to common food antigens

Heavy Metal Testing

Purpose:

Assess toxic load contribution to anxiety

Levels of mercury, lead, arsenic, cadmium that can impair neurological function

Genetic Testing

Purpose:

Identify genetic factors affecting anxiety regulation

MTHFR, COMT, 5-HTTLPR, and other polymorphisms affecting neurotransmitter function

Treatment Protocol

Our Integrative Approach

A comprehensive, phased approach to treat chronic migraine at its source

1
Phase 1

Comprehensive assessment and root cause identification

Comprehensive assessment and root cause identification

Complete medical, psychiatric, and trauma history,Advanced laboratory testing (blood, urine, stool),Neurotransmitter and adrenal function assessment,Validated anxiety rating scales (GAD-7, Hamilton Anxiety Scale),Cognitive pattern assessment,Sleep quality evaluation

2
Phase 2

Restore neurotransmitter balance and reduce acute symptoms

Restore neurotransmitter balance and reduce acute symptoms

Click to expand

3
Phase 3

Address underlying drivers of anxiety

Address underlying drivers of anxiety

Click to expand

4
Phase 4

Build long-term resilience and prevent relapse

Build long-term resilience and prevent relapse

Click to expand

5
Phase 5

Sustain gains and optimize function

Sustain gains and optimize function

Click to expand

Diet & Lifestyle

Supporting Your Treatment

Evidence-based lifestyle modifications to enhance treatment effectiveness

Success Metrics

What Success Looks Like

GAD-7 score reduction to <10 (mild range)

Ability to confront avoided situations without catastrophic outcomes

Restored sleep quality (7-8 hours, feeling refreshed)

Return to full social and occupational functioning

Stable mood throughout the day without anxiety spikes

Reduced physical symptoms (palpitations, tension headaches, digestive issues)

Improved cognitive clarity and concentration

Maintained progress through life stressors

Common Questions

Frequently Asked Questions

Expertise Behind This Guide

Evidence-Based Information

Dr. Hafeel Sevdeer - DHA Licensed Integrative and Functional Medicine Physician with advanced training in anxiety disorders, stress physiology, and the gut-brain axis. Specialist in treating treatment-resistant anxiety using comprehensive functional medicine approaches combined with evidence-based psychotherapy.

References

  1. 1. Bandelow B, Michaelis S. Epidemiology of anxiety disorders in the 21st century. Dialogues Clin Neurosci. 2015.
  2. 2. Craske MG, Stein MB. Anxiety. Lancet. 2016.
  3. 3. Hoge EA, Chen MM, Wright E, et al. Meditation and magnesium for anxiety. J Clin Psychopharmacol. 2022.
  4. 4. Lakhan SE, Vieira KF. Nutritional and herbal supplements for anxiety and anxiety-related disorders. Nutr J. 2010.
  5. 5. National Institute for Health and Care Excellence. Generalised anxiety disorder and panic disorder in adults: management. NICE Guidelines. 2020.
  6. 6. Strawn JR, Geracioti L, Rajdev N, et al. Pharmacotherapy for generalized anxiety disorder in adults. Expert Opin Pharmacother. 2018.
  7. 7. Walker FR, Hodes M. The neurobiology of stress and anxiety. Handb Clin Neurol. 2020.
  8. 8. Yehuda R, Seckl J. Minireview: Stress-related cortisol dysfunction and brain aging. Brain Res. 2011.
  9. 9. Bystritsky A, Khalsa SS, Cameron ME, et al. Current diagnosis and treatment of anxiety disorders. P T. 2013.
  10. 10. Hofmann SG, Asnaani A, Vonk IJ, et al. The efficacy of cognitive behavioral therapy. Cogn Behav Ther. 2012.

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