+971 56 274 1787WhatsApp
psychiatric-behavioral-health ConditionNeurological

Insomnia & Sleep Disorders

"Taking more than 30 minutes to fall asleep most nights"

15+
Days/Month
50-70%
Medication Overuse
2-3x
Stroke Risk
Reversible
With Treatment
Understanding Your Condition

What is Chronic Migraine?

Insomnia is a chronic sleep disorder characterized by persistent difficulty falling asleep, staying asleep, or achieving restorative sleep despite adequate opportunity and circumstances. It involves dysregulation of the sleep-wake cycle, often driven by hyperarousal of the central nervous system, circadian rhythm disruption, or underlying physiological imbalances. This condition affects 10-30% of adults worldwide and significantly impacts physical health, cognitive function, and emotional wellbeing when left untreated.

Healthy Function

What your body should do

Healthy sleep follows a precise circadian rhythm governed by the suprachiasmatic nucleus (SCN) in the hypothalamus. The sleep-wake cycle operates on a 24-hour clock regulated by light exposure, with melatonin secretion beginning 2-3 hours before bedtime in response to dim light. Sleep architecture cycles through NREM stages 1-3 (light to deep sleep) and REM sleep every 90-110 minutes, with 4-6 complete cycles per night. Deep NREM sleep (slow-wave sleep) facilitates physical restoration, cellular repair, and glymphatic clearance of metabolic waste from the brain. REM sleep supports emotional regulation, memory consolidation, and cognitive processing. A healthy adult requires 7-9 hours of sleep with 15-20% deep sleep and 20-25% REM sleep for optimal function.

When Things Go Wrong

Signs of chronification

  • Pain threshold lowers over time
  • More frequent attacks
  • Brain stays in alert mode
  • Medication stops working
Development Process

How This Develops

Understanding the biological mechanisms helps us target the root cause

Point 1

Understanding the mechanism helps us target the root cause rather than just treating symptoms.

Symptom Manifestations

Recognizing All Symptoms

Chronic migraine affects multiple systems. Understanding your symptoms helps us identify the underlying mechanisms.

Physical Symptoms

12 symptoms

  • Difficulty falling asleep (sleep onset insomnia)
  • Frequent nighttime awakenings (sleep maintenance insomnia)
  • Early morning awakening with inability to return to sleep
  • Non-restorative sleep despite adequate time in bed
  • Daytime fatigue and exhaustion
  • Morning headaches or grogginess
  • Muscle tension and physical restlessness
  • Gastrointestinal discomfort at night
  • Increased pain sensitivity
  • Frequent urination during the night (nocturia)
  • Restless legs or periodic limb movements
  • Teeth grinding (bruxism) during sleep

Cognitive Symptoms

10 symptoms

  • Racing thoughts at bedtime
  • Difficulty concentrating during the day
  • Memory problems and forgetfulness
  • Brain fog and mental sluggishness
  • Reduced problem-solving ability
  • Impaired decision-making
  • Slower reaction times
  • Difficulty learning new information
  • Reduced creativity and mental flexibility
  • Preoccupation with sleep and worry about not sleeping

Emotional Symptoms

10 symptoms

  • Anxiety about sleep and bedtime
  • Irritability and mood swings
  • Depression and low mood
  • Heightened emotional reactivity
  • Feeling overwhelmed by daily tasks
  • Social withdrawal due to fatigue
  • Decreased motivation and interest
  • Sense of hopelessness about recovery
  • Panic attacks at night
  • Emotional dysregulation and volatility

Metabolic Symptoms

11 symptoms

  • Weight gain and difficulty losing weight
  • Increased appetite and carbohydrate cravings
  • Blood sugar instability
  • Insulin resistance
  • Elevated cortisol levels
  • Hormonal imbalances (cortisol, melatonin, growth hormone)
  • Reduced leptin and elevated ghrelin (hunger hormones)
  • Impaired glucose tolerance
  • Elevated inflammatory markers
  • Compromised immune function
  • Thyroid hormone conversion impairment
Commonly Associated

Conditions That Occur Together

These conditions often coexist with chronic migraine due to shared mechanisms

Related Condition

Anxiety Disorders

Hyperarousal from anxiety activates the sympathetic nervous system and HPA axis, elevating cortisol and norepinephrine that prevent sleep onset and maintenance; creates anticipatory anxiety about sleep itself

Related Condition

Depression

Depression disrupts circadian rhythm and REM sleep architecture; insomnia and depression share neurotransmitter imbalances (serotonin, dopamine) and create a bidirectional worsening cycle

Related Condition

Chronic Pain

Pain signals activate the reticular activating system, maintaining cortical arousal; inflammatory cytokines from pain disrupt sleep centers; poor sleep lowers pain threshold, creating vicious cycle

Related Condition

Adrenal Dysfunction

Elevated evening cortisol from HPA axis dysregulation prevents melatonin production and sleep onset; disrupted cortisol rhythm causes early morning awakening with inability to return to sleep

Related Condition

Gut Dysbiosis

Intestinal permeability increases systemic inflammation (IL-6, TNF-alpha) that activates brain wake centers; gut bacteria produce 90% of serotonin, affecting melatonin synthesis; SIBO causes nighttime discomfort

Related Condition

Thyroid Dysfunction

Hyperthyroidism increases metabolic rate and sympathetic tone, preventing sleep; hypothyroidism causes sleep apnea and non-restorative sleep; both disrupt thermoregulation needed for sleep

Related Condition

Sleep Apnea

Recurrent breathing disruptions cause microarousals that fragment sleep architecture; oxygen desaturation triggers sympathetic activation; creates sleep maintenance insomnia even after apnea treatment

Related Condition

Blood Sugar Dysregulation

Nocturnal hypoglycemia triggers cortisol and adrenaline surges that wake the brain; insulin resistance affects orexin neurons that regulate wakefulness; poor sleep worsens glucose metabolism

Differential Diagnoses

Conditions to Rule Out

These conditions can present similarly but have distinct features

Condition

Sleep Apnea

Overlapping

Non-restorative sleep, daytime fatigue, frequent awakenings

Key Difference

Sleep apnea shows witnessed breathing pauses, loud snoring, gasping; confirmed by elevated AHI on polysomnography; oxygen desaturation events present

Condition

Restless Legs Syndrome (RLS)

Overlapping

Difficulty falling asleep, frequent awakenings, daytime fatigue

Key Difference

RLS causes irresistible urge to move legs with uncomfortable sensations; symptoms worsen at rest and improve with movement; low ferritin often present

Condition

Circadian Rhythm Sleep-Wake Disorder

Overlapping

Difficulty falling asleep, daytime sleepiness, non-restorative sleep

Key Difference

CRSWD shows normal sleep quality when allowed to sleep at preferred times; delayed sleep phase causes inability to sleep until very late; melatonin rhythm shifted

Condition

Narcolepsy

Overlapping

Excessive daytime sleepiness, disrupted nighttime sleep

Key Difference

Narcolepsy includes cataplexy, sleep paralysis, hypnagogic hallucinations; MSLT shows rapid sleep onset and REM periods; different pathophysiology (orexin deficiency)

Condition

Chronic Fatigue Syndrome

Overlapping

Persistent fatigue, unrefreshing sleep, cognitive difficulties

Key Difference

CFS features post-exertional malaise and widespread pain; primary complaint is fatigue rather than sleep disturbance; sleep studies may show normal architecture

Condition

Medication-Induced Insomnia

Overlapping

Difficulty falling or staying asleep

Key Difference

Clear temporal relationship to medication initiation; common culprits include SSRIs, steroids, beta-agonists, decongestants, stimulants; improves when medication discontinued

Condition

Substance-Induced Sleep Disorder

Overlapping

Sleep onset or maintenance difficulties

Key Difference

Associated with alcohol, caffeine, nicotine, or drug use; alcohol causes initial sedation followed by fragmented sleep; caffeine has long half-life affecting sleep

Root Causes

What's Driving Your Migraines

Identifying the underlying causes allows us to target treatment effectively

1

Chronic Stress and HPA Axis Dysregulation

45% - Persistent activation of stress response elevates cortisol and norepinephrine, creating physiological hyperarousal that prevents sleep onset and maintenance

Comprehensive stress inventory, salivary cortisol rhythm testing, ACTH levels, assessment of life stressors and coping mechanisms

2

Circadian Rhythm Disruption

35% - Irregular sleep schedules, shift work, jet lag, and blue light exposure suppress melatonin production and misalign the biological clock

Sleep diary analysis, melatonin level testing, assessment of light exposure patterns, evaluation of work schedule and travel history

3

Gut Dysbiosis and Leaky Gut

25% - Intestinal permeability increases inflammation affecting sleep centers; gut bacteria imbalances reduce serotonin and melatonin production

Comprehensive stool analysis, zonulin testing, food sensitivity panels, SIBO breath testing, assessment of digestive symptoms

4

Nutrient Deficiencies

30% - Deficiencies in magnesium, vitamin D, B-vitamins, iron, and zinc impair neurotransmitter synthesis and sleep regulation

Full micronutrient panel, RBC magnesium, ferritin, 25-OH vitamin D, organic acids test for B-vitamin status

5

Blood Sugar Dysregulation

20% - Nocturnal hypoglycemia triggers stress hormones that wake the brain; insulin resistance affects sleep-wake regulating neurons

Continuous glucose monitoring, fasting insulin, HbA1c, glucose tolerance test, assessment of nighttime eating patterns

6

Neurotransmitter Imbalances

25% - Altered GABA, serotonin, dopamine, and orexin levels disrupt the sleep-wake switch and prevent transitions to sleep states

Organic acids testing, neurotransmitter metabolite assessment, amino acid panel, evaluation of mood and cognitive symptoms

7

Environmental and Lifestyle Factors

30% - Poor sleep hygiene, bedroom environment, caffeine and alcohol use, irregular schedules, and lack of physical activity

Detailed sleep hygiene questionnaire, bedroom environment evaluation, substance use history, exercise patterns, screen time assessment

8

Hormonal Changes

20% - Menopause, perimenopause, menstrual cycle fluctuations, and andropause cause temperature dysregulation and hormonal shifts that disrupt sleep

Hormone panel (estrogen, progesterone, testosterone, FSH, LH), assessment of menopausal symptoms, menstrual cycle tracking

9

Chronic Infections and Inflammation

15% - Low-grade infections (EBV, Lyme) and chronic inflammation elevate cytokines that activate wake-promoting brain regions

Viral panels, inflammatory markers (CRP, IL-6, TNF-alpha), assessment of infection history, evaluation of inflammatory symptoms

Lab Assessment

Key Laboratory Markers

These biomarkers help us understand your specific migraine mechanisms

Test
Normal Range
Optimal Range
Clinical Significance
Melatonin (Evening)
Normal:10-60 pg/mL pg/mL
Optimal:30-50 pg/mL pg/mL
Low evening melatonin indicates circadian disruption; optimal levels promote sleep onset
Cortisol (Evening/Salivary)
Normal:2-10 ng/mL ng/mL
Optimal:<2 ng/mL ng/mL
Elevated evening cortisol indicates HPA axis dysregulation and hyperarousal preventing sleep
Cortisol Awakening Response (CAR)
Normal:50-75% increase from waking percentage
Optimal:50-75% increase, then steady decline percentage
Blunted or exaggerated CAR indicates HPA axis dysfunction affecting sleep-wake regulation
Vitamin D (25-OH)
Normal:30-100 ng/mL ng/mL
Optimal:50-80 ng/mL ng/mL
Vitamin D deficiency strongly associated with sleep disorders and poor sleep quality
Magnesium (RBC)
Normal:4.2-6.8 mg/dL mg/dL
Optimal:6.0-6.8 mg/dL mg/dL
Magnesium regulates GABA receptors and melatonin production; deficiency causes sleep fragmentation
Iron (Ferritin)
Normal:15-150 ng/mL (women), 30-400 ng/mL (men) ng/mL
Optimal:70-100 ng/mL ng/mL
Low ferritin linked to restless legs syndrome and sleep fragmentation; essential for dopamine synthesis
Thyroid Stimulating Hormone (TSH)
Normal:0.45-4.5 mIU/L mIU/L
Optimal:1.0-2.0 mIU/L mIU/L
Both hyper and hypothyroidism disrupt sleep architecture; optimal range ensures metabolic stability
Hs-CRP (Inflammation)
Normal:<3 mg/L mg/L
Optimal:<1 mg/L mg/L
Elevated inflammation markers correlate with poor sleep quality and insomnia severity
Cost of Waiting

What Happens If Left Untreated

Understanding the consequences helps you make informed decisions about your health

Cardiovascular Disease

2-5 years

Chronic insomnia increases risk of hypertension by 3-4x, coronary artery disease by 45%, and stroke by 54% through sympathetic overactivation and inflammation

Type 2 Diabetes and Metabolic Syndrome

1-5 years

Sleep deprivation impairs glucose metabolism and insulin sensitivity; increases risk of diabetes by 28% and promotes weight gain through appetite hormone disruption

Cognitive Decline and Dementia

5-15 years

Poor sleep prevents glymphatic clearance of amyloid-beta and tau proteins; chronic insomnia increases Alzheimer's risk by 53% and accelerates cognitive aging

Mental Health Disorders

6-24 months

Insomnia increases depression risk by 10x and anxiety disorders by 17x; creates bidirectional worsening cycle that becomes increasingly difficult to break

Immune Dysfunction

Ongoing

Sleep deprivation reduces natural killer cell activity by 70%, impairs antibody response to vaccines, and increases susceptibility to infections and cancer

Accidents and Injuries

Immediate and ongoing

Sleep deprivation causes impairment equivalent to blood alcohol of 0.05-0.10%; responsible for 100,000+ motor vehicle accidents and 1,550 deaths annually

Chronic Pain Syndromes

1-3 years

Poor sleep lowers pain threshold and increases pain sensitivity; insomnia sufferers 2x more likely to develop chronic pain conditions

Hormonal Disruption and Infertility

1-5 years

Sleep deprivation suppresses growth hormone, testosterone, and reproductive hormones; reduces fertility and accelerates aging processes

Time Matters

Don't wait for symptoms to worsen. Early intervention leads to better outcomes.

Diagnostic Approach

How is Chronic Migraine Diagnosed?

Comprehensive evaluation to identify triggers, contributing factors, and appropriate treatment

Comprehensive Sleep Assessment

Purpose:

Detailed evaluation of sleep patterns, quality, and contributing factors

Sleep efficiency, sleep latency, wake after sleep onset, sleep architecture, identification of sleep disruptors

Salivary Cortisol Rhythm (4-point)

Purpose:

Assess HPA axis function and circadian cortisol pattern

Elevated evening cortisol, blunted CAR, or flattened curve indicating hyperarousal and stress dysregulation

Melatonin Profile

Purpose:

Evaluate circadian rhythm and melatonin production

Low evening melatonin indicating circadian disruption; delayed melatonin onset suggesting phase delay

Comprehensive Blood Panel

Purpose:

Identify metabolic, hormonal, and inflammatory contributors

Thyroid function, blood sugar markers, inflammatory markers, complete metabolic panel, CBC

Micronutrient Testing

Purpose:

Identify deficiencies affecting sleep neurochemistry

Magnesium, vitamin D, iron/ferritin, B-vitamins, zinc levels critical for sleep regulation

Organic Acids Test

Purpose:

Evaluate mitochondrial function, neurotransmitter metabolism, and nutrient status

B-vitamin status, neurotransmitter metabolites, oxidative stress markers, gut microbial metabolites

Gut Microbiome Analysis

Purpose:

Assess gut health impact on sleep through gut-brain axis

Dysbiosis patterns, inflammation markers, bacterial imbalances affecting neurotransmitter production

Continuous Glucose Monitoring (CGM)

Purpose:

Identify nocturnal blood sugar fluctuations disrupting sleep

Nighttime hypoglycemia events, glucose variability, relationship between meals and sleep quality

Hormone Panel

Purpose:

Evaluate sex hormones and their impact on sleep

Estrogen, progesterone, testosterone, DHEA-S levels affecting sleep quality and thermoregulation

Inflammatory Markers

Purpose:

Assess systemic inflammation affecting sleep centers

Hs-CRP, IL-6, TNF-alpha levels that activate wake-promoting brain regions

Treatment Protocol

Our Integrative Approach

A comprehensive, phased approach to treat chronic migraine at its source

1
Phase 1

Comprehensive evaluation and immediate sleep environment optimization

Comprehensive evaluation and immediate sleep environment optimization

2
Phase 2

Calm hyperarousal and establish healthy sleep pressure

Calm hyperarousal and establish healthy sleep pressure

Click to expand

3
Phase 3

Address underlying physiological imbalances

Address underlying physiological imbalances

Click to expand

4
Phase 4

Consolidate gains and prevent relapse

Consolidate gains and prevent relapse

Click to expand

Diet & Lifestyle

Supporting Your Treatment

Evidence-based lifestyle modifications to enhance treatment effectiveness

Success Metrics

What Success Looks Like

Sleep onset latency <20 minutes

Sleep efficiency >85% (time asleep/time in bed)

Wake after sleep onset <30 minutes total

7-8 hours of restorative sleep nightly

Morning energy levels 7+/10 without caffeine

Elimination of daytime fatigue and sleepiness

Normalized cortisol rhythm on salivary testing

Improved mood and cognitive function

Reduced or eliminated sleep medication dependence

Sustained sleep quality during stress periods

Resolution of nighttime awakenings

Improved metabolic markers (glucose, inflammation)

Common Questions

Frequently Asked Questions

Expertise Behind This Guide

Evidence-Based Information

Dr. Hafeel Afsar, DHA Licensed Integrative Medicine practitioner with advanced training in sleep medicine, functional endocrinology, and neurological health. Specializes in identifying root causes of sleep disorders through comprehensive assessment of hormonal, metabolic, and neurological factors. Expertise in circadian rhythm restoration, HPA axis regulation, and integrative approaches to chronic insomnia that address the whole person rather than merely suppressing symptoms.

References

  1. 1. 1. Morin CM, et al. Insomnia: Nature, Diagnosis, and Treatment. Sleep Med Clin. 2013;8(3):389-398. doi:10.1016/j.jsmc.2013.05.009
  2. 2. 2. Baglioni C, et al. Insomnia as a predictor of depression: A meta-analytic evaluation of longitudinal epidemiological studies. J Affect Disord. 2011;135(1-3):10-19. doi:10.1016/j.jad.2011.01.003
  3. 3. 3. Cappuccio FP, et al. Sleep duration and all-cause mortality: a systematic review and meta-analysis of prospective studies. Sleep. 2010;33(5):585-592. doi:10.1093/sleep/33.5.585
  4. 4. 4. Besedovsky L, et al. The Sleep-Immune Crosstalk in Health and Disease. Physiol Rev. 2019;99(3):1325-1380. doi:10.1152/physrev.00010.2018
  5. 5. 5. Walker MP. Why We Sleep: Unlocking the Power of Sleep and Dreams. Scribner; 2017.
  6. 6. 6. Irish LA, et al. The role of sleep hygiene in promoting public health: A review of empirical evidence. Sleep Med Rev. 2015;22:23-36. doi:10.1016/j.smrv.2014.10.001
  7. 7. 7. Trauer JM, et al. Cognitive Behavioral Therapy for Chronic Insomnia: A Systematic Review and Meta-analysis. Ann Intern Med. 2015;163(3):191-204. doi:10.7326/M14-2841
  8. 8. 8. Zhai L, et al. Association between vitamin D and sleep disorders: A systematic review and meta-analysis. Nutrients. 2020;12(12):3684. doi:10.3390/nu12123684

Ready to Find Relief from Chronic Migraines?

Our integrative approach has helped hundreds of patients find lasting relief from chronic migraines. Schedule your comprehensive assessment today.

500+ Patients Treated
15+ Years Experience
Integrative Approach