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psychiatric-behavioral-health ConditionNeurological

OCD & PTSD

"Intrusive, unwanted thoughts or memories that feel impossible to control or dismiss"

15+
Days/Month
50-70%
Medication Overuse
2-3x
Stroke Risk
Reversible
With Treatment
Understanding Your Condition

What is Chronic Migraine?

Obsessive-Compulsive Disorder (OCD) and Post-Traumatic Stress Disorder (PTSD) are distinct but often comorbid neuropsychiatric conditions characterized by dysregulated fear processing, intrusive mental phenomena, and maladaptive behavioral patterns. OCD involves recurrent obsessions (intrusive thoughts, images, or urges) and compulsions (repetitive behaviors performed to neutralize obsessions or prevent feared outcomes). PTSD develops following exposure to actual or threatened death, serious injury, or sexual violence, featuring intrusive re-experiencing, avoidance, negative alterations in cognition and mood, and marked alterations in arousal and reactivity. Both conditions involve amygdala hyperactivity, hippocampal dysfunction, prefrontal cortex impairment, and HPA axis dysregulation.

Healthy Function

What your body should do

A healthy stress response system maintains appropriate threat detection through the amygdala while the prefrontal cortex provides top-down regulation to distinguish real from perceived danger. The hippocampus accurately contextualizes memories in time and place. The HPA axis responds to genuine threats with appropriate cortisol release, followed by efficient recovery and return to homeostasis. Fear extinction occurs naturally when threats pass. Intrusive thoughts are recognized as mental noise and dismissed without distress. Sleep architecture supports memory consolidation and emotional processing. The autonomic nervous system maintains balance between sympathetic activation and parasympathetic restoration.

When Things Go Wrong

Signs of chronification

  • Pain threshold lowers over time
  • More frequent attacks
  • Brain stays in alert mode
  • Medication stops working
Development Process

How This Develops

Understanding the biological mechanisms helps us target the root cause

Point 1

Understanding the mechanism helps us target the root cause rather than just treating symptoms.

Symptom Manifestations

Recognizing All Symptoms

Chronic migraine affects multiple systems. Understanding your symptoms helps us identify the underlying mechanisms.

Physical Symptoms

12 symptoms

  • Racing heart, palpitations, and chest tightness during triggers
  • Shortness of breath and hyperventilation episodes
  • Muscle tension, especially in neck, shoulders, and jaw
  • Gastrointestinal distress: nausea, diarrhea, IBS flare-ups
  • Chronic fatigue despite adequate sleep
  • Insomnia and nightmares disrupting sleep architecture
  • Startle response hyperreactivity
  • Sweating and trembling during anxiety episodes
  • Headaches and migraines
  • Dizziness and feeling faint
  • Chronic pain syndromes (fibromyalgia comorbidity)
  • Immune dysfunction and frequent infections

Cognitive Symptoms

12 symptoms

  • Intrusive, unwanted thoughts or memories
  • Rumination and obsessive worry loops
  • Difficulty concentrating and memory problems
  • Catastrophic thinking and worst-case scenario fixation
  • Mental checking and reviewing behaviors
  • Thought-action fusion (believing thoughts equal actions)
  • Hypervigilance and constant threat scanning
  • Dissociation and feeling detached from reality
  • Negative self-beliefs and distorted cognitions
  • Decision paralysis and overthinking
  • Flashbacks with impaired reality testing
  • Impaired executive function and planning

Emotional Symptoms

12 symptoms

  • Persistent fear and sense of impending doom
  • Guilt and shame about symptoms or trauma
  • Emotional numbing and restricted affect
  • Irritability and anger outbursts
  • Anhedonia and loss of interest in activities
  • Overwhelming anxiety and panic
  • Depression and hopelessness
  • Self-blame and worthlessness
  • Trust issues and relationship difficulties
  • Feeling permanently damaged or broken
  • Isolation and withdrawal from support systems
  • Suicidal ideation in severe cases
Commonly Associated

Conditions That Occur Together

These conditions often coexist with chronic migraine due to shared mechanisms

Related Condition

Depression and Mood Disorders

Shared neurobiological pathways involving serotonin depletion, HPA axis dysregulation, and inflammatory processes; up to 50% of PTSD patients and 40% of OCD patients experience comorbid depression

Related Condition

Anxiety Disorders

Generalized anxiety, panic disorder, and social anxiety share amygdala hyperactivity and fear circuitry dysfunction; often predate or coexist with OCD/PTSD

Related Condition

Substance Use Disorders

Self-medication to manage distressing symptoms; 40-60% of PTSD patients develop substance abuse; temporarily reduces amygdala activation but worsens long-term outcomes

Related Condition

Sleep Disorders

Sleep deprivation impairs prefrontal cortex regulation while increasing amygdala reactivity; nightmares disrupt REM sleep critical for fear extinction

Related Condition

Chronic Pain Syndromes

Central sensitization and shared neuroinflammatory processes; trauma and chronic stress alter pain processing pathways; fibromyalgia comorbidity in 20-30%

Related Condition

Autoimmune Conditions

Chronic inflammation and immune dysregulation from PTSD/OCD increase autoimmune risk; shared genetic vulnerabilities and environmental triggers

Related Condition

Cardiovascular Disease

Chronic sympathetic activation and cortisol elevation damage vascular endothelium; PTSD associated with 2x increased cardiovascular risk

Related Condition

Gut-Brain Axis Dysfunction

Trauma and chronic stress alter gut microbiome, reduce vagal tone, and increase intestinal permeability; gut inflammation exacerbates neuroinflammation

Related Condition

Dissociative Disorders

Severe trauma can fragment identity and memory processing; dissociation serves as psychological escape when fight/flight is impossible

Related Condition

Eating Disorders

Trauma history common in eating disorders; control behaviors around food parallel OCD compulsions; body image distortion linked to trauma

Differential Diagnoses

Conditions to Rule Out

These conditions can present similarly but have distinct features

Condition

OCD (Obsessive-Compulsive Disorder)

Overlapping

Intrusive thoughts, anxiety, repetitive behaviors

Key Difference

Ego-dystonic obsessions with specific compulsions performed to neutralize anxiety; insight often preserved but resistance is difficult; no trauma history required

Condition

PTSD (Post-Traumatic Stress Disorder)

Overlapping

Intrusive memories, avoidance, hypervigilance

Key Difference

Direct link to traumatic event; re-experiencing phenomena; negative alterations in cognition/mood; symptoms persist >1 month after trauma

Condition

Complex PTSD (C-PTSD)

Overlapping

PTSD symptoms plus emotional dysregulation, negative self-concept

Key Difference

Results from prolonged, repeated trauma (childhood abuse, captivity); includes disturbances in self-organization: affect dysregulation, negative self-concept, disturbed relationships

Condition

Generalized Anxiety Disorder

Overlapping

Excessive worry, hypervigilance, physical anxiety symptoms

Key Difference

Worry is diffuse and not tied to specific obsessions or trauma; no compulsions or re-experiencing phenomena; content of worry shifts across life domains

Condition

Panic Disorder

Overlapping

Panic attacks, anticipatory anxiety, avoidance

Key Difference

Panic attacks are spontaneous and not triggered by specific obsessions or trauma cues; fear focuses on panic sensations themselves

Condition

Social Anxiety Disorder

Overlapping

Avoidance, fear of judgment, physical anxiety

Key Difference

Fear specifically centers on social evaluation and embarrassment; no intrusive obsessions or trauma-related re-experiencing

Condition

Body Dysmorphic Disorder

Overlapping

Repetitive checking, intrusive thoughts about appearance

Key Difference

Preoccupation specifically with perceived defects in appearance; compulsions focus on appearance-checking or concealing

Condition

Hoarding Disorder

Overlapping

Difficulty discarding, repetitive acquisition behaviors

Key Difference

Previously considered OCD subtype; distress centers on parting with possessions rather than intrusive obsessions; living spaces become unusable

Condition

Trichotillomania/Excoriation

Overlapping

Repetitive behaviors, tension reduction

Key Difference

Body-focused repetitive behaviors without preceding obsessions; behaviors provide gratification or relief rather than preventing feared outcomes

Condition

Adjustment Disorder

Overlapping

Emotional distress following stressor, functional impairment

Key Difference

Stressor can be any life change (not just trauma); symptoms are less severe and resolve within 6 months of stressor removal

Condition

Acute Stress Disorder

Overlapping

PTSD-like symptoms following trauma

Key Difference

Symptoms occur immediately after trauma and resolve within 1 month; if persistent beyond 1 month, diagnosis becomes PTSD

Condition

Psychotic Disorders

Overlapping

Intrusive thoughts, fear, behavioral changes

Key Difference

OCD obsessions are recognized as own thoughts (not inserted); no formal thought disorder; reality testing preserved outside of specific obsessional fears

Root Causes

What's Driving Your Migraines

Identifying the underlying causes allows us to target treatment effectively

1

Trauma Exposure (PTSD-specific)

90% - Direct exposure to actual or threatened death, serious injury, or sexual violence is required for PTSD diagnosis; trauma type, severity, and duration affect risk

Comprehensive trauma history including childhood adversity, combat exposure, assault, accidents, natural disasters; assess peritraumatic dissociation

2

Genetic Predisposition

30-40% - Family history increases risk 2-4x; serotonin transporter gene (5-HTTLPR), COMT, and BDNF polymorphisms implicated

Family psychiatric history; genetic testing for relevant polymorphisms affecting neurotransmitter metabolism and stress response

3

Childhood Adversity and Attachment

50-60% for PTSD, 30% for OCD - Early life stress alters developing stress response systems and creates vulnerability

ACE (Adverse Childhood Experiences) score; attachment style assessment; developmental history including neglect, abuse, or household dysfunction

4

HPA Axis Dysregulation

40% - Chronic or severe stress dysregulates hypothalamic-pituitary-adrenal axis function

Cortisol testing (morning, evening, diurnal curves); DHEA-S; ACTH levels; evaluate stress history and coping capacity

5

Neurotransmitter Imbalance

35% - Serotonin depletion, dopamine dysregulation, and GABA deficiency impair fear processing and behavioral inhibition

Urinary neurotransmitter panels; amino acid testing; methylation status (MTHFR, homocysteine); response to SSRI trial

6

Neuroinflammation

25% - Elevated pro-inflammatory cytokines affect neuroplasticity, neurotransmitter metabolism, and blood-brain barrier integrity

Inflammatory markers (CRP, IL-6, TNF-alpha); gut permeability testing; infectious disease screening; autoimmune markers

7

Gut Microbiome Dysbiosis

20% - Altered gut bacteria reduce GABA and serotonin production, increase systemic inflammation, and impair vagus nerve signaling

Comprehensive stool analysis; SIBO breath testing; assessment of antibiotic history, diet, and digestive symptoms

8

Nutrient Deficiencies

20% - B vitamins, magnesium, zinc, omega-3s, and vitamin D are essential for neurotransmitter synthesis and neuronal health

Comprehensive micronutrient panel; RBC magnesium; omega-3 index; vitamin D levels; dietary assessment

9

Brain Structure and Function

25% - Reduced hippocampal and prefrontal cortex volume; amygdala hyperactivity; default mode network dysfunction

Neuropsychological testing; qEEG brain mapping; structural MRI if indicated; functional connectivity assessment

10

Infectious and Toxic Contributors

15% - PANDAS/PANS (pediatric autoimmune), Lyme disease, mold toxicity, and heavy metals can trigger or exacerbate symptoms

ASO and anti-DNase B titers; Lyme and co-infection testing; mycotoxin panel; heavy metal screening

11

Cognitive and Learning Factors

30% - Thought-action fusion, intolerance of uncertainty, and anxiety sensitivity maintain OCD; maladaptive cognitions maintain PTSD

Validated questionnaires (OBQ, IIQ, ASI); cognitive assessment; trauma-related cognitions inventory

Lab Assessment

Key Laboratory Markers

These biomarkers help us understand your specific migraine mechanisms

Test
Normal Range
Optimal Range
Clinical Significance
Cortisol (Morning)
Normal:5-25 mcg/dL mcg/dL
Optimal:8-14 mcg/dL mcg/dL
HPA axis function; PTSD often shows elevated or blunted morning cortisol
Cortisol (Evening)
Normal:<10 mcg/dL mcg/dL
Optimal:<5 mcg/dL mcg/dL
Flattened diurnal curve common in PTSD; impaired recovery from daily stress
DHEA-S
Normal:150-350 mcg/dL mcg/dL
Optimal:200-300 mcg/dL mcg/dL
Adrenal reserve; often depleted in chronic PTSD and OCD
Serotonin
Normal:50-200 ng/mL ng/mL
Optimal:100-150 ng/mL ng/mL
Mood and impulse regulation; deficiency linked to OCD and PTSD severity
C-Reactive Protein (hs-CRP)
Normal:<3.0 mg/L mg/L
Optimal:<1.0 mg/L mg/L
Systemic inflammation; elevated in PTSD and associated with symptom severity
Homocysteine
Normal:<15 umol/L umol/L
Optimal:<10 umol/L umol/L
Methylation status; elevated levels impair neurotransmitter synthesis
Vitamin D
Normal:30-100 ng/mL ng/mL
Optimal:50-70 ng/mL ng/mL
Neuroprotection and mood regulation; deficiency associated with both conditions
Magnesium (RBC)
Normal:4.0-6.4 mg/dL mg/dL
Optimal:5.0-6.0 mg/dL mg/dL
Nervous system relaxation; deficiency exacerbates hyperarousal symptoms
B12
Normal:200-900 pg/mL pg/mL
Optimal:500-800 pg/mL pg/mL
Neurological function and methylation; deficiency affects cognitive symptoms
TSH
Normal:0.4-4.0 mIU/L mIU/L
Optimal:1.0-2.0 mIU/L mIU/L
Thyroid function; dysregulation can mimic or worsen anxiety symptoms
Omega-3 Index
Normal:>4% percentage
Optimal:8-12% percentage
Neuroinflammation marker; low levels associated with mood disorders
8-OHdG (Oxidative Stress)
Normal:<500 ng/mg creatinine ng/mg creatinine
Optimal:<300 ng/mg creatinine ng/mg creatinine
DNA oxidative damage marker; elevated in chronic stress states
Cost of Waiting

What Happens If Left Untreated

Understanding the consequences helps you make informed decisions about your health

Chronic Neurobiological Dysregulation

Months to years

Untreated OCD/PTSD causes progressive changes in brain structure and function, including hippocampal atrophy and prefrontal cortex impairment, making recovery increasingly difficult

Treatment Resistance Development

Years

Longer duration of untreated illness predicts poorer response to treatment; neural pathways become more entrenched; may require more intensive interventions

Substance Abuse and Dependence

Often within first year of symptom onset

Self-medication with alcohol, benzodiazepines, cannabis, or other substances leads to addiction; dual diagnosis complicates treatment and worsens outcomes

Major Depression Development

Months to years

60-80% of untreated PTSD and 50% of untreated OCD develop comorbid depression; suicide risk increases significantly with combined conditions

Cardiovascular Disease

Years to decades

Chronic sympathetic activation and inflammation increase risk of hypertension, coronary artery disease, and stroke; PTSD associated with 2x cardiovascular mortality

Metabolic Syndrome and Diabetes

Years

Chronic cortisol dysregulation promotes insulin resistance, weight gain, and metabolic dysfunction

Autoimmune Disease

Years

Chronic inflammation and immune dysregulation increase risk of autoimmune conditions including rheumatoid arthritis, lupus, and thyroid disease

Relationship and Social Deterioration

Progressive

Avoidance behaviors, emotional numbing, and symptom preoccupation damage intimate relationships; social isolation increases; divorce rates elevated

Occupational Disability

Months to years

Concentration impairment, avoidance, and symptom severity reduce work performance; many patients become unable to work; significant economic impact

Suicide Risk

Ongoing risk

OCD carries 10x increased suicide risk; PTSD associated with significant suicide risk, especially with comorbid depression; requires vigilant monitoring

Physical Health Comorbidities

Years

Chronic pain, gastrointestinal disorders, respiratory conditions, and immune dysfunction become increasingly prevalent

Quality of Life Degradation

Immediate and progressive

Symptoms consume increasing time and energy; joy and fulfillment diminish; life becomes organized around symptoms rather than values and goals

Time Matters

Don't wait for symptoms to worsen. Early intervention leads to better outcomes.

Diagnostic Approach

How is Chronic Migraine Diagnosed?

Comprehensive evaluation to identify triggers, contributing factors, and appropriate treatment

Comprehensive Psychiatric Evaluation

Purpose:

Establish diagnosis and assess severity

Structured clinical interview for DSM-5 criteria; differential diagnosis; comorbidity assessment; suicide risk evaluation

Yale-Brown Obsessive Compulsive Scale (Y-BOCS)

Purpose:

Assess OCD symptom severity

Quantified obsessions, compulsions, avoidance, and insight; tracks treatment progress; scores >16 indicate clinically significant symptoms

Clinician-Administered PTSD Scale (CAPS-5)

Purpose:

Gold standard PTSD assessment

Frequency and intensity of all PTSD symptom clusters; diagnostic confirmation; severity rating; treatment response monitoring

PCL-5 (PTSD Checklist)

Purpose:

Self-report PTSD screening

Symptom presence and severity; score >31-33 suggests probable PTSD; useful for tracking changes

Comprehensive Blood Panel

Purpose:

Rule out medical causes and assess biological contributors

CBC, CMP, thyroid function, inflammatory markers, cortisol, DHEA-S, vitamin D, B12, magnesium, homocysteine

Neurotransmitter Panel

Purpose:

Assess neurochemical status

Urinary levels of serotonin, dopamine, norepinephrine, GABA, glutamate; guides targeted amino acid therapy

Adrenal Function Testing

Purpose:

Evaluate HPA axis status

Diurnal cortisol curves, DHEA-S, cortisol awakening response; reveals dysregulation patterns

Stool Microbiome Analysis

Purpose:

Assess gut-brain axis contribution

Bacterial diversity, pathogenic organisms, inflammation markers, SCFA production, leaky gut indicators

Nutritional and Micronutrient Testing

Purpose:

Identify deficiencies affecting brain function

Comprehensive vitamin, mineral, amino acid, and fatty acid status; omega-3 index

Genetic Testing

Purpose:

Identify genetic factors affecting treatment

MTHFR, COMT, 5-HTTLPR, BDNF Val66Met; informs medication selection and nutrient therapy

qEEG Brain Mapping

Purpose:

Assess brain electrical activity patterns

Abnormalities in frontal lobe function, amygdala connectivity, and fear circuitry; guides neurofeedback if indicated

Trauma and Attachment Assessment

Purpose:

Comprehensive trauma history and impact

ACE score, trauma type/severity, attachment style, dissociation levels, complex PTSD features

Toxic and Infectious Screening

Purpose:

Rule out environmental contributors

Heavy metals, mycotoxins, Lyme disease, PANDAS/PANS markers when clinically indicated

Treatment Protocol

Our Integrative Approach

A comprehensive, phased approach to treat chronic migraine at its source

1
Phase 1

Thorough diagnostic evaluation, safety planning, and acute symptom management

Thorough diagnostic evaluation, safety planning, and acute symptom management

Complete psychiatric and trauma-informed medical history,Structured diagnostic interviews (CAPS-5, Y-BOCS, SCID),Comprehensive laboratory testing and biological assessment,Suicide risk assessment and safety planning,Sleep stabilization and hygiene optimization,Psychoeducation about OCD/PTSD neurobiology,Establish therapeutic alliance and treatment expectations,Begin foundational self-regulation skills,Address acute substance use if present,Coordinate care with existing providers

2
Phase 2

Restore neurochemical balance and reduce symptom severity

Restore neurochemical balance and reduce symptom severity

Click to expand

3
Phase 3

Process traumatic memories and rewire fear responses

Process traumatic memories and rewire fear responses

Click to expand

4
Phase 4

Consolidate gains and build long-term resilience

Consolidate gains and build long-term resilience

Click to expand

5
Phase 5

Sustain recovery and optimize quality of life

Sustain recovery and optimize quality of life

Click to expand

Diet & Lifestyle

Supporting Your Treatment

Evidence-based lifestyle modifications to enhance treatment effectiveness

Success Metrics

What Success Looks Like

Y-BOCS score reduction to <16 (subclinical range) for OCD

PCL-5 score reduction to <31-33 for PTSD

Ability to experience intrusive thoughts/trauma triggers without significant distress

Elimination of compulsive behaviors or safety behaviors

Restored sleep quality with minimal nightmares

Return to full occupational and social functioning

Stable mood without significant anxiety or hyperarousal episodes

Improved relationships and social connection

Reduced physiological reactivity to triggers

Ability to tolerate uncertainty and distress

Values-based living rather than symptom-driven existence

Resilience in face of life stressors without relapse

Common Questions

Frequently Asked Questions

Expertise Behind This Guide

Evidence-Based Information

Dr. Hafeel Sevdeer - DHA Licensed Integrative and Functional Medicine Physician with advanced training in trauma-informed care, OCD treatment, and the neurobiology of stress. Specialist in combining evidence-based psychotherapy with functional medicine approaches for treatment-resistant OCD and PTSD. Certified in trauma-focused interventions with expertise in the gut-brain axis, HPA axis regulation, and neuroinflammatory contributors to mental health conditions.

References

  1. 1. American Psychiatric Association. Diagnostic and Statistical Manual of Mental Disorders, 5th Edition (DSM-5). 2013.
  2. 2. Stein DJ, Costa DLC, Lochner C, et al. Obsessive-compulsive disorder. Nat Rev Dis Primers. 2019.
  3. 3. Kessler RC, Aguilar-Gaxiola S, Alonso J, et al. Trauma and PTSD in the WHO World Mental Health Surveys. Eur J Psychotraumatol. 2017.
  4. 4. Foa EB, Yadin E, Lichner TK. Exposure and Response (Ritual) Prevention for Obsessive-Compulsive Disorder: Therapist Guide. 2012.
  5. 5. Shapiro F. The role of eye movement desensitization and reprocessing (EMDR) therapy in medicine. Perm J. 2014.
  6. 6. Bandelow B, Baldwin D, Abelli M, et al. Biological markers for anxiety disorders, OCD and PTSD. Eur Arch Psychiatry Clin Neurosci. 2017.
  7. 7. Yehuda R, Hoge CW, McFarlane AC, et al. Post-traumatic stress disorder. Nat Rev Dis Primers. 2015.
  8. 8. Abercrombie ED, Jacobs BL. Single-unit response of noradrenergic neurons in the locus coeruleus of freely moving cats. I. Acutely presented stressful and nonstressful stimuli. J Neurosci. 1987.
  9. 9. Bremner JD, Randall P, Vermetten E, et al. Magnetic resonance imaging-based measurement of hippocampal volume in posttraumatic stress disorder. Arch Gen Psychiatry. 1997.
  10. 10. Foster JA, Rinaman L, Cryan JF. Stress & the gut-brain axis: Regulation by the microbiome. Neurobiol Stress. 2017.
  11. 11. Sarris J, Murphy J, Mischoulon D, et al. Adjunctive nutraceuticals for depression: A systematic review. J Affect Disord. 2016.
  12. 12. Wilkinson ST, Ballard ED, Bloch MH, et al. The effect of a single dose of intravenous ketamine on suicidal ideation. JAMA Psychiatry. 2018.
  13. 13. Hofmann SG, Asnaani A, Vonk IJ, et al. The efficacy of cognitive behavioral therapy: A review of meta-analyses. Cognit Ther Res. 2012.
  14. 14. Neria Y, Nandi A, Galea S. Post-traumatic stress disorder following disasters: A systematic review. Psychol Med. 2008.
  15. 15. Pittenger C, Bloch MH, Williams K. Glutamate abnormalities in obsessive compulsive disorder. Neurobiol Dis. 2011.

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