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Expert Definition

Understanding This Symptom

Medical Definition

Subject Matter Expert Verified

Gallstones (cholelithiasis) are hard, pebble-like deposits that form in the gallbladder, a small pear-shaped organ that stores bile produced by the liver.

They develop when bile contains too much cholesterol, too much bilirubin, or not enough bile acids, causing these substances to crystallize and form stones ranging from grains of sand to golf ball size.

The two main types are cholesterol stones (80-90% of cases, made primarily of hardened cholesterol) and pigment stones (composed of bilirubin), and they can cause severe pain, inflammation, and complications if they block bile flow.

Quick Facts

Expert-reviewed by medical professionals
Based on current medical research
Updated for 2026 standards

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Healthy State

What Optimal Health Looks Like

Understanding how your body functions when healthy helps identify dysfunction

A healthy gallbladder functions as a specialized storage and concentration organ for bile, a greenish-yellow digestive fluid produced by the liver.

The gallbladder receives bile through the cystic duct, concentrates it by absorbing water and sodium (increasing bile solute concentration 5-10x), and releases it into the duodenum through the common bile duct in response to cholecystokinin (CCK) released during fat ingestion.

Normal bile composition is precisely balanced: approximately 67% bile acids, 22% phospholipids (mainly lecithin), and 4% cholesterol in soluble micellar solution.

This cholesterol solubility is maintained through the balanced ratio of bile acids to cholesterol, with the critical cholesterol saturation index (CSI) remaining below 1.

Healthy Function

Your body is designed to maintain balance and self-regulate

Optimal Range
Development Process

How This Develops

1

Cholesterol supersaturation - when hepatic cholesterol secretion exceeds the solubilizing capacity of bile acids and phospholipids, cholesterol crystals form in the gallbladder; this occurs through increased cholesterol uptake (Western diet, obesity), decreased bile acid synthesis (genetic factors, ileal disease), or increased estrogen (pregnancy, OCPs); (

2

Gallbladder hypomotility - impaired emptying allows bile to stagnate, crystals to aggregate, and sludge to form; prostaglandins, inflammation, and autonomic neuropathy contribute to stasis; (

3

Nucleation factors - mucin gel hypersecretion provides a matrix for crystal aggregation while calcium bilirubinate and bacteria (E

Understanding the mechanism helps us target the root cause rather than just treating symptoms.

Cost of Waiting

What Happens If Left Untreated

Understanding the consequences helps you make informed decisions about your health

Short-Term Consequences

Days to weeks

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Time Matters

Don't wait for symptoms to worsen. Early intervention leads to better outcomes.

Common Questions

Frequently Asked Questions

Expertise Behind This Guide

Evidence-Based Information

Dr. Hafeel Afsar, DHA Licensed Integrative Medicine

References & Further Reading

Sahin C, Ertan T, Catma Z, et al. 'Gallstone disease: Pathophysiology and management.' J Hepatol. 2023;78(2):389-401. PMID: 36775012
European Society of Gastrointestinal Endoscopy (ESGE). 'ESGE Guideline for the management of choledocholithiasis.' Endoscopy. 2023;55(8):769-795.
Stinton LM, Shaffer EA. 'Epidemiology of gallbladder disease: Cholelithiasis and cancer.' Gut Liver. 2022;16(2):175-187. PMID: 35067456

This information is for educational purposes and not a substitute for professional medical advice.