+971 56 274 1787WhatsApp
Symptom

Health Information

Comprehensive undefined information and treatment options at Healers Clinic Dubai.

"You are not alone in this journey."

15,000+ Patients
DHA Licensed
95% Success Rate
27+ Years
Expert Definition

Understanding This Symptom

Medical Definition

Subject Matter Expert Verified

Multiple sclerosis (MS) is a chronic autoimmune demyelinating disease of the central nervous system where the immune system mistakenly attacks the myelin sheath surrounding nerve fibers, disrupting electrical signal transmission between the brain and body.

This demyelination leads to scar tissue (sclerosis) formation along damaged nerve pathways, causing a wide range of neurological symptoms that vary greatly between individuals.

MS typically presents in young adults (20-40 years old), with women affected 2-3 times more frequently than men.

Quick Facts

Expert-reviewed by medical professionals
Based on current medical research
Updated for 2026 standards

Need More Information?

Our experts can provide personalized guidance

Book Consultation
Healthy State

What Optimal Health Looks Like

Understanding how your body functions when healthy helps identify dysfunction

In a healthy central nervous system, oligodendrocytes produce and maintain the myelin sheath - a fatty insulating layer that wraps around nerve axons, enabling rapid electrical signal transmission (saltatory conduction).

Myelin allows nerve impulses to travel up to 100 times faster than unmyelinated fibers.

The blood-brain barrier (BBB) protects the CNS from peripheral immune cells.

Axons remain intact and healthy, with efficient communication between neurons.

Healthy Function

Your body is designed to maintain balance and self-regulate

Optimal Range
Development Process

How This Develops

1

Genetic predisposition - HLA-DRB

2

Environmental triggers - Epstein-Barr virus infection, low vitamin D, smoking, gut microbiome dysbiosis

3

Blood-brain barrier disruption - Peripheral autoreactive T-cells (CD

4

cross the BBB, aided by matrix metalloproteinases (MMPs)

5

Inflammatory demyelination - Activated T-cells release pro-inflammatory cytokines (IFN-gamma, TNF-alpha, IL-

6

that recruit B-cells and macrophages

7

Myelin destruction - Macrophages and microglia phagocytose myelin sheaths

8

Oligodendrocyte death - Direct immune attack and inflammatory cytokines kill oligodendrocytes, impairing remyelination

9

Axonal degeneration - Secondary axonal loss correlates with permanent disability

10

Lesion formation - Discrete areas of demyelination (plaques) form in white matter, particularly periventricular regions, brainstem, spinal cord, and cerebellum

11

Progressive disease - In chronic stages, neurodegeneration predominates with cortical demyelination and brain atrophy

Understanding the mechanism helps us target the root cause rather than just treating symptoms.

Cost of Waiting

What Happens If Left Untreated

Understanding the consequences helps you make informed decisions about your health

Short-Term Consequences

Days to weeks

  • [object Object] | [object Object] | [object Object]

Time Matters

Don't wait for symptoms to worsen. Early intervention leads to better outcomes.

Common Questions

Frequently Asked Questions

Expertise Behind This Guide

Evidence-Based Information

Dr. Hafeel Afsar, DHA Licensed Integrative and Functional Medicine Practitioner Specialization: Neurological disorders, autoimmune conditions, neurodegenerative disease Qualifications: Board-certified in Integrative Medicine, Advanced Neuroimmunology Training Experience: 15+ years treating Multiple Sclerosis and neuroinflammatory conditions with integrative protocols

References & Further Reading

Filippi M, Bar-Or A, Piehl F, et al. Multiple Sclerosis. Nat Rev Dis Primers. 2018;4(1):43. PMID: 30410042 - Comprehensive review of MS pathophysiology, diagnosis, and treatment.
Thompson AJ, Banwell BL, Barkhof F, et al. Diagnosis of Multiple Sclerosis: 2017 Revisions of the McDonald Criteria. Lancet Neurol. 2018;17(2):162-173. PMID: 29275977 - Updated diagnostic criteria for MS.
Hauser SL, Bar-Or A, Comi G, et al. Ocrelizumab versus Interferon Beta-1a in Relapsing Multiple Sclerosis. N Engl J Med. 2017;376(3):221-234. PMID: 28002688 - Landmark trial demonstrating efficacy of anti-CD20 therapy in MS.
Bjornevik K, Cortese M, Healy BC, et al. Longitudinal analysis reveals high prevalence of Epstein-Barr virus associated with multiple sclerosis. Science. 2022;375(6578):296-301. PMID: 35025605 - Evidence establishing EBV as primary trigger for MS.
Ascherio A, Munger KL. Environmental risk factors for multiple sclerosis. Part I: the role of infection. Ann Neurol. 2007;61(4):288-299. PMID: 17444504 - Review of infectious triggers including Epstein-Barr virus.
Munger KL, Levin LI, Hollis BW, et al. Serum 25-hydroxyvitamin D levels and risk of multiple sclerosis. JAMA. 2006;296(23):2832-2838. PMID: 17179460 - Evidence linking vitamin D deficiency to MS risk.

This information is for educational purposes and not a substitute for professional medical advice.